Insulin resistance and visceral obesity can affect metabolic health. Understanding how these factors interact is essential for preventing and treating metabolic diseases including type 2 diabetes, cardiovascular disease, and others.
Insulin Resistance: Definition: Insulin resistance arises when cells become less insulin-responsive. Pancreatic insulin regulates blood sugar (glucose).
Hyperinsulinemia: The pancreas produces more insulin to compensate for insulin resistance, causing high blood insulin levels.
High blood sugar levels occur from glucose dysregulation, which prevents cells from taking up glucose more efficiently despite higher insulin. This can cause prediabetes and type 2 diabetes.
Inflammation and Insulin Resistance: Inflammatory Response: Insulin resistance and visceral obesity cause chronic low-grade inflammation.
Metabolic Health Impacts: - Type 2 Diabetes: Insulin resistance can initiate type 2 diabetes. High blood sugar and insulin dysfunction describe this disease.
Interventions and Management: Lifestyle changes: Eating well and exercising regularly are essential. Working out and losing weight enhance insulin sensitivity and resistance.
Insulin resistance and belly obesity require lifestyle changes, diet changes, and even pharmacological therapies. Personalised metabolic health management requires regular health checks and interactions with healthcare providers.